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Shingles Nerve Treatment

Shingles Not a skin disease but a nerve disease.

Shingles Not a skin disease but a nerve disease. — Source: 대상포진 피부 발진 — 한쪽 피부분절을 따라 띠 모양으로 나타난다

Source : 대상포진 피부 발진 — 한쪽 피부분절을 따라 띠 모양으로 나타난다

Reactivation of herpes virus lying dormant in a ganglion provokes severe inflammation there.
Early in the course, before the rash appears in the affected dermatome, diagnosis can be difficult.
What matters most is early treatment that minimizes nerve damage.

Shingles Early treatmentmatters

Severe accompanying pain or an extensive rash means the associated nerve damage is severe, and the risk of progressing to postherpetic neuralgia is high.
In such cases, additional medication and nerve treatment given early in the course can do as much as possible to prevent progression to postherpetic neuralgia.
If the window for treatment is missed Progresses to postherpetic neuralgia in 50% of patients in their fifties and 80% of those in their eightiesand you may be left suffering severe pain.

At high risk of progressing to postherpetic neuralgia High-risk group

  • Advanced age

    Patients aged 50 and over, or older

  • Severe early pain

    Severe pain early in the course

  • Chronic condition

    Those with long-standing chronic conditions (diabetes, liver or kidney disease)

  • Autoimmune disease

    Those with an autoimmune condition such as rheumatoid arthritis

  • Cranial nerve involvement

    Shingles involving the cranial nerves (eye, ear, face)

In acute shingles Treatment method?

In acute shingles Treatment method? — Source: 대상포진 신경뿌리치료 실제 사진 — 본원 의료진 시행

Source : 대상포진 신경뿌리치료 실제 사진 — 본원 의료진 시행

In the acute phase, the following three are carried out together. The order is the order of priority.

  1. STEP 01

    Prompt administration of antiviral medication

    Whether tablet or injection, prompt administration after onset is best.

  2. STEP 02

    Nerve treatment

    The aim is to inject directly into the inflamed, damaged spinal ganglion to reduce the intensity of inflammation and calm nerve hyperexcitability.

  3. STEP 03

    Appropriate control of pain intensity

    Enduring severe pain is itself harmful to proper recovery of nerve function. Adequate pain control through appropriate prescribing by a specialist, combined with supportive therapy, can reduce the extent of nerve damage.

※ This is general medical information provided for understanding only. Whether and how to treat is determined by a specialist after examination, based on the individual patient's condition.

Advanced Equipment

  • SIEMENS3T AI MRIWe add AI imaging (Deep Resolve) to a proven Siemens 3T MRI to examine neurological conditions faster and more clearly.
  • MRACerebrovascular and carotid MRAIt accurately evaluates the degree of atherosclerosis, stenosis, structural abnormality, and the presence of aneurysm in the vessels of the brain and neck.
  • Carotid USCarotid UltrasoundCarotid wall thickness and plaque are examined to assess stroke risk.
  • NCS / EMGNerve conduction studiesNerves in the arms, legs, and face are safely stimulated electrically and the responses recorded, establishing whether peripheral nerve damage is present and how severe it is.
  • EPEvoked potential testBy safely stimulating the visual and auditory pathways electrically, damage to the cranial nerves can be identified.
  • EEGAI digital EEGBeyond the conventional identification of epileptiform waveforms, quantitative EEG with the latest AI software aids in assessing brain function in headache, anxiety, and depression.
  • VNGVideo nystagmographyBy recording and analyzing involuntary eye movement on video, this test is useful for distinguishing dizziness disorders such as BPPV and Meniere's disease.
  • HRV / SSRAutonomic Function TestEquipment that objectively assesses the balance between the body's sympathetic and parasympathetic nerves.
  • PWV / ABIArterial elasticity testingBy measuring vessel elasticity and the degree of atherosclerosis, it readily assesses the risk of circulatory disturbance and cardiovascular or cerebrovascular disease.
  • Wearable ECG/BPWearable cardiac function testingA micro-sensor continuously analyzes blood pressure and ECG over 24 hours to identify cardiac dysfunction capable of causing a stroke.
  • SNSB / CERADNeuropsychological testingIt distinguishes simple forgetfulness from dementia and objectively evaluates cognitive function including memory.
  • LABBlood and genetic testingWe identify risk factors for cardiovascular and cerebrovascular disease, including dyslipidemia, and look for high-risk dementia genes in advance so that prevention can begin.